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Levels of reactive oxygen species (ROS) produced during the stress impair mitochondrial DNA and result in dysfunctional electron transport chains and reduced ATP synthesis

This effect may be partly due to weight loss, though other mechanisms may also contribute to cardiovascular benefits demonstrated in large-scale studies

Pathogenesis An increased number of melanocytes within the basal layer of the epidermis leads to an increased production of melanin, resulting in hyperpigmented macules

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Given that GLP-1 therapy builds gradually, each weeks delay can blunt patient enthusiasm and worsen clinical inertia