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UCP also acts on the abnormal calcium transport between the endoplasmic reticulum and mitochondria, causing mitochondrial calcium overload through RyR2 and VDAC, which leads to weakened stress tolerance [67]

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Cellular proliferation of various cell types such as endothelial, epithelial, or stromal cells is one of the customary hallmarks exhibited by endometriosis which promotes enhanced survival and implantation of these cells at ectopic sites [55,56,57]

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