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Notably, a significant portion of albumin in the blood lacks the C-terminal L585 residue, ranging from 3 to 15% in healthy individuals and up to 93% in patients with pancreatitis or other pancreatic disorders 60,61,62

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Notably, loss-of-function mutations in ABCG2 (e.g., Q141K) impair intestinal urate excretion, thereby significantly increasing the risk of gout (Kawaguchi et al., 2021

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