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A drug facing LOE in a therapeutic class with branded successors will have payers actively managing the transition in ways that accelerate new-patient generic uptake

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In addition, it has long been known that GSH is capable of forming disulfide bonds with cysteine residues of proteins, and the relevance of this mechanism (S-glutathionylation) in regulation of protein function has been well documented in a number of research fields

By Kim Severson If you need help staying positive in the midst of heat waves and out-of-office disruptions, start with these tips