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Hum Mol Genet 8:14511460 Reaume A, Elliott J, Hoffman E et al (1996) Motor neurons in Cu/Zn superoxide dismutase-deficient mice develop normally but exhibit enhanced cell death after axonal injury

Second, given that the activation of GSR and TXNRD1 are involved in ROS elimination, if the LCS3-triggered lethality of VRK2-KO PC cells could be attributed to inhibiting the activity of both GSR and TXNRD1, then the level of ROS generated by LCS3 should have been much higher than those produced by the treatment of the TXNRD1 inhibitor auranofin

A sick cell, on the other hand, does not compensate and the value stays positive

Precautions involve avoiding direct eye contact and discontinuing if severe irritation occurs

T.LeissnerP.SaliouA.et al (2023)