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In our current studies we tested if inhibition of antibody production was drug-specific or whether other widely used over-the counter NSAIDs would also interfere with antibody synthesis
The experiment demonstrates the potential of IGF-I treatment of catabolic states and especially the value of modified forms of growth factors that bind weakly to IGF-binding proteins. In another study in the Journal of Developmental Cell , IGF-1 LR3 was shown to help prevent muscle wasting and stimulate muscle growth through the activation of the IGF1R/IRS1/PI3K/Akt pathway
We postulated that 5FU treatment may promote an enhanced dependency on mitochondrial metabolism, and, in turn, mitochondrial metabolism may dictate sensitivity to 5FU
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