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[Google Scholar] 17.Prescott L.F., Donovan J.W., Jarvie D.R., Proudfoot A.T

Furthermore, molecular hydrogen may help preserve NADPH levels by: Protecting glucose-6-phosphate dehydrogenase (G6PD), the rate-limiting enzyme in the pentose phosphate pathway that generates NADPH Reducing overall cellular oxidative status, decreasing NADPH consumption by other systems Preserving mitochondrial function, supporting energy metabolism that indirectly maintains NADPH production This conservation of NADPH availability ensures that glutathione reductase has adequate cofactor supply to perform its glutathione recycling function, especially during periods of increased oxidative challenge

Therefore, the uncertainty resulting from some prospective studies is partially resolved, because elevated circulating homocysteine is a major independent causative risk factor for cardiovascular disease

Culina, S

Deficiency of G6PD affects HMP Shunt in the lens that leads to shortening of ribose availability for the synthesis of nucleic acid and decreases the renewal of lens proteins