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A buildup in AD has the potential to damage synapses and cause neuroinflammation by triggering astroglia and microglia cells [4, 52, 53]
Although this study focused on the risk of pancreatitis, the cardiovascular, renal, and metabolic benefits of GLP-1 RAs, which are well-documented in the literature, reinforce the relevance of this drug class and suggest that their continued use might be favorable
LMP-1, triptolide and UA can mediate their effects through ERK1/2 activation, while lactoferricin suppresses TLR2/4