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Since slow-acting cell death processes can proceed following ototoxin removal (Owens et al., To verify DASPEI scores, fish from the same treatment groups were fixed and immunohistochemically processed with anti-parvalbumin (Millipore) to visualize hair cells (see Coffin et al., 3).These results indicate that Dihexa only modulates targets responsible for the acute phase of aminoglycoside ototoxicity

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Glutamine metabolism inhibition, using agents such as CB-839, has shown potential to suppress TCA cycle-mediated energy production and disrupt redox homeostasis, particularly in CRC subtypes resistant to 5-FU and oxaliplatin [145]

IGF-1 levels vary significantly by age and sex, so results should be interpreted using the correct reference range