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Given the high numbers of microbial cells and genes 18 , it is now evident that the gut microbiome can impact glucose and energy homeostasis via numerous mechanisms, including both impacting host gutbrain signaling and initiating direct communication to the brain via microbe-derived metabolites 19

Clinical isolates show considerable genomic diversity, including chromosomal duplications and non-classical mutations, complicating treatment strategies (Priest et al., 2022

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Our APAP model [10] is simple because it does not include the processing of GSH in the mitochondria that has been shown to be fundamental for liver cell death in the presence of APAP [36]