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Since Uch-L1 and UBB+1 exert opposite effects on A processing, metabolism and polyubiquitin chain formation, pharmacological modulation of UBB+1 or Uch-L1 levels in the brain may be a potential therapeutic strategy for AD

Given its energetic requirements, it should be no surprise that researchers are increasingly making connections between the brain and mitochondrial health (mitochondria are power horse organelles found in most cells and are responsible for cellular respiration and energy production)

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