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By contrast, preclinical in vivo and in vitro studies both indicate that incretin receptor agonism can attenuate fibrogenic signalling in models of kidney disorders, and thus the positive clinical trial data on kidney function outcome measures may, in part, be mediated by inhibition of fibrosis

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We hypothesized that L-carnitine, by providing a constant production of ATP molecules, can stimulate PCr formation, leading to a low optimal efficacious dose of creatine [14]