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The progression of CP is clinically observable, starting with acinar cell dysfunction, followed by beta-cell dysfunction, and lastly, a decrease in alpha-cell function which signifies the end stage of the disease

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421 Immunotherapeutic approaches face further barriers due to the immunosuppressive TME

Arhalofenate Acid Inhibits Monosodium Urate crystal-induced Inflammatory Responses through Activation of AMP-Activated Protein Kinase (AMPK) Signaling