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While the long-term consequences of GLP-1 receptor agonism are now emerging, similar long-term studies will be required to investigate the safety of new co-agonists in the brain and cardiovascular system, and their potential for promoting cell proliferation [39]

Conclusions: Our results reveal a significant association between PI3K/AKT signaling pathway inhibition and HACD and offer potential therapeutic targets for developing novel treatment strategies for HACD

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