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Rodent and human beta-cells have reduced transcriptional and protein levels of cytosolic copper/zinc (Cu/Zn) SOD1, mitochondrial manganese (Mn) SOD2, catalase, and GPx, which can result in exacerbated levels of superoxide, H 2 O 2 , hydroxyl radical, and peroxynitrite that are implicated in beta-cell death in T1D (5761)

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