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All authors have made a significant contribution to this study and have approved the final manuscript

Once carnitine metabolism is disrupted, it is not only a reflection of impaired renal function, but also may promote the occurrence and progression of DKD through mitochondrial energy dysfunction, oxidative stress and inflammatory responses

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It has been proposed that depletion of hepatic GSH, particularly mitochondrial GSH, is one of the early changes associated with chronic alcohol consumption and is a critical contributor to ALD pathogenesis 11

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