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Targeted metabolomics analysis revealed that PHP-DPS@INS effectively normalized the elevated level of mitochondrial proinflammatory metabolite fumarate in an in vitro hypertonic model of DED, thereby reducing the levels of key inflammatory factors (IL-1, IL-6, and TNF-)

Vitamin D also suppressed development of pulmonary emphysema, epithelialmesenchymal transition (EMT), and fibrogenesis, all of which are associated with the development of COPD and are exacerbated by toxicant exposure [24, 25]