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Mitochondrial ROS production and oxidative mtDNA damage is increased during reoxygenation in the GPX1 knockout mice, in addition to structural abnormalities in cardiac mitochondria and myocytes, suggesting GPX1 may play an important role in protecting cardiac mitochondria from reoxygenation damage in vivo

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The 9-h and 24-h DHA treatments similarly persisted in altering ribosome synthesis, protein-DNA complex assembly, proteasome activity, proteasomal protein catabolism, immune responses, cytoplasmic translation, peptide biosynthesis, and ribosome biogenesis

Behav Brain Res 208:545552

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