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[24] indicated that allicin protected cardiac function and prevented the development of cardiac hypertrophy through ROS-dependent mechanisms involving multiple intracellular signalling, such as ERK1/2, JNK1/2, and AKT signalling pathways

1996 Phase I First clinical study Dorr et al

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In Simple Terms Used in research studying tissue repair and cellular migration pathways Often included in tendon, ligament, connective tissue, and wound-healing models Supports comparison of repair-signaling activity under controlled lab conditions Quick Summary: BPC-157 is supplied as a 10mg lyophilized research compound