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Another study determined that GLP-1 could restore insulin secretion via modulation of actin polymerization, independent of a recently identified role for the mammalian target of rapamycin complex 2 (mTORC2) in actin modulation and insulin sensitivity 9

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The glucose is oxidized in the transition metal-dependent reaction (TMDR) to the ene-diol anion, which is transformed into ketoaldehydes and superoxide anions