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Zhao, F., Gao, X., Ge, X., Cui, J

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Abstract Mitochondrial defects result in dysregulation of metabolomics and energy homeostasis that are detected in upper motor neurons (UMNs) with TDP-43 pathology, a pathology that is predominantly present in both familial and sporadic cases of amyotrophic lateral sclerosis (ALS)

In MS, a recent systematic review and meta-analysis assessed folate metabolism and epigenetic implications

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