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Recent reviews have highlighted potential immunomodulatory effects across diverse autoimmune contexts, suggesting that the observed anti-inflammatory properties may extend beyond metabolic improvement (77)

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The GLP-1 analogs, semaglutide and liraglutide, delay the progression of atherosclerosis by regulating inflammatory pathways in low-density lipoprotein receptor-deficient (LDLr -/- ) and apolipoprotein E-deficient (ApoE -/- ) mice [16]

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