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In animal studies, exenatide (and its analogue exendin-4) increased microbes tied to better metabolism (47)

In particular, Gokce and Sekmen (2025) demonstrated that the knockout of AtGLR3.4.1 and AtGLR3.4.2 disrupted Reactive Oxygen Species (ROS) homeostasis and altered the transcription of mitochondrial stress-related genes such as Cytochrome c oxidase subunit 5B (COX5B) , uncoupling protein-related oxidase (UPOX) and uncoupling protein 1 (UCP1)

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The presence of eNOS results in greater dilation of the blood vessel and less stress on the vascular wall