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This reorganization is essential for forming structures like lamellipodia and filopodia, which are the "feet" that cells use to crawl and migrate

4.3.15, CompOmics) employing four algorithms (Comet, Tide, MetaMorpheus and Sage)

Conversely, cartilage-specific overexpression of GPX4 via transgenic technology, or restoration of its activity using small molecule drugs such as certain natural compounds or ferroptosis inhibitors, can significantly delay cartilage degeneration, improve subchondral bone sclerosis and osteophyte formation, and alleviate synovitis in model animals (Xiao et al., 2024), which directly confirms the protective role of GPX4 in OA progression from a gain-of-function perspective

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