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Numerous studies have demonstrated that the inhibition of inflammatory cytokines mediated by NF-B and HIF-1, such as AP and curcumin, significantly alleviates cerebral edema (45, 46)

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The fact that mutations in the hereditary hemochromatosis protein (HFE) have not been found to increase the risk of AML (89, 90) may also indicate that de novo AML development is not induced by systemic iron overload