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This temporal lag may provide a clear explanation for the delayed onset (ranging from hours to days post-activity) and the persistent nature (with the cycle remaining resistant to spontaneous resolution) of PEM symptoms (22, 23)

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Similarly, ferroptosis-mediated cell death that results in the release of 8-hydroxylamine (8-OHG) activates the STING1-dependent inflammatory pathway in surrounding macrophages and promotes M2 polarization [115]