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While both peptides have been studied for tissue-regenerative properties, they appear to function through different mechanisms: BPC-157 may work primarily through growth factor-mediated pathways and nitric oxide systems
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doi: 10.1053/jhep.2003.50351
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Therefore, by inhibiting canonical wingless-related integration site (Wnt)/-catenin signaling, the augmentation of bone production by lutein may need the reduction of sclerostin expression
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