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Importantly, some of these structural abnormalities persist despite levothyroxine replacement, reflecting the inadequacy of systemic therapy in restoring intracerebral hormone balance (79, 80)

Ruemmele FM, Beaulieu JF, Dionne S et al (2002) Lipopolysaccharide modulation of normal enterocyte turnover by toll-like receptors is mediated by endogenously produced tumour necrosis factor alpha

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