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As for the mechanisms that might drive this important modification of the metabolic setting in hyperinsulinemic obese PCOS patients, it has to be stated that the four complementary substances we administered each have the ability, singly, to positively modulate insulin sensitivity, reducing IR in PCOS patients (12, 30-32)

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This finding agrees with the literature whereby the role of quinone redox metabolism is an ambiguous source of toxicity, with evidence suggesting that redox cycling requires supra-pharmacological concentrations of doxorubicin to generate substantial ROS in tissues and cells 10

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