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doi:10.1016/j.biocel.2012.08.016, PMID: 22939972 Zeevalk GD, Manzino L, Sonsalla PK, Bernard LP (2007) Characterization of intracellular elevation of glutathione (GSH) with glutathione monoethyl ester and GSH in brain and neuronal cultures: relevance to Parkinsons disease

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Schomburg L (2012) Selenium, selenoproteins and the thyroid gland: interactions in health and disease

However, like any intravenous treatment, it should be performed in a clinical setting with appropriate screening

GYY4137 attenuates LPS/ATP-induced mitochondrial dysfunctions by inhibiting the NF-kB signaling pathway We next sought to determine the mechanism by which GYY4137 attenuates LPS/ATP elicited mitochondrial dysfunctions