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1,2,33 Excessive ROS levels may cause serious oxidative damage, which can be reduced by an antioxidant system formed by SODs, catalases, MAPKS, SIRTs, GSH, vitamin C and E

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Men carrying high-risk variants in these genes often experience superior outcomes with dual-compound GLP-1 therapy because both semaglutide and tirzepatide address these pathways simultaneously

identified the hypermethylation of the CpG island in the death receptor 3 (DR3) gene in RASFs, resulting in reduced expression of the DR3 protein [32]

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