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[5] The researchers also note that IR and IGF1R act as identical portals to the regulation of gene expression, with differences between insulin and IGF-1 effects due to a modulation of the amplitude of the signal created by the specific ligand-receptor interaction. The IGF-1R is referred to as the physiologic receptor due to its potential higher affinity (approximately 100 times higher) for IGF-1 as compared to the insulin receptor

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The paper proposes, a final infusion rate of 12.5 mg/kg/h for a 32 g overdose, and a final infusion rate of 18.75 mg/kg/h for a 48 g overdose

Hence, there are at least two distinct mechanisms for regulating IGF-1R activity in response to a cells mechanical loading state

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