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Impairments to the BBB disrupt the brain's microenvironment homeostasis, ultimately resulting in neuronal injury and functional anomalies, a phenomenon extensively documented in neurodegenerative disease studies [1]

These results showed that SLC25A45 is one of the cold-induced mitochondrial solute carriers

assess the effect of L-carnitine on VLDL cholesterol level and no significant correlation was found.[] However, L-carnitine may change metabolic pathways from esterification in the liver and change TG synthesis to ALC, which can reduce the synthesis of TG and VLDL cholesterol.[] Also, catabolism of VLDL is reduced in individuals with diabetes, which is due to decreased activity of LPL in adipose tissue leading to augmented levels of TG in individuals with diabetes

Several explanations exist, and they are not mutually exclusive

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