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And aspirin inhibits its own activity by acetylating the K384, K394, and K414 residues of cGAS

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GLUCOSE-DEPENDENT INSULIN STIMULATION (Pancreatic Beta Cells): Activates GLP-1 receptors on pancreatic beta cells stimulates insulin secretion ONLY when blood glucose is elevated This glucose-dependence is a critical safety advantage: minimal risk of hypoglycaemia (low blood sugar) when used alone Promotes beta cell proliferation and survival potentially preserving insulin-secreting capacity over time 2

Journal of interventional cardiology