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This molecular aberration results in a significantly reduced production of proinflammatory cytokines, type I interferons, and chemokines subsequent to pathogenic challenge [182, 183]

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Despite a relatively low percentage of patients with preexisting CVD compared with previous CVOTs, the primary composite MACE outcome (including death from unknown causes) occurred in 12% of the dulaglutide group and 13.4% of the placebo group