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This increase in DAT function stems at least in part from augmented DAT surface expression, pointing to DAT trafficking as the underlying mechanism by which GLP-1R signaling and AA levels controls DA homeostasis in the LS, as depicted by our schematic model (Supplementary Figure 5)

The primary endpoint was to identify changes in plasma metabolic profiles following the GF-MKD intervention

[DOI] [PMC free article] [PubMed] [Google Scholar] 47.Cosentino F., Grant P.J., Aboyans V., et al

This molecular event triggers formation of multi-protein signalling complexes containing paxillin, talin, and vinculin, detectable through co-immunoprecipitation assays and proximity ligation microscopy techniques