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These effects were mediated through enhanced AMP-activated protein kinase signaling and diminished PI3K/Akt signaling, indicating that glutamine supports intestinal barrier function via modulation of specific intracellular pathways (110, 118)

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Additional analyses were performed by gender and age to investigate changes from baseline in HbA 1c and body weight, adverse events (AEs), and hypoglycemia, as defined by the American Diabetes Association (ADA) [17]