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This suppression is independent of plasma cholesterol changes and BW reduction through regulating markers of plaque instability and inflammation including those linked to plaque hemorrhage (CD163), matrix turnover (matrix metalloproteinase (MMP)-3, MMP-13), cholesterol metabolism (prostaglandin I2 synthase (PTGIS), ATP-binding cassette transporter 1 (ABCA1)), leukocyte recruitment (C-C motif chemokine ligand 2 (CCL2), osteopontin (OPN), interleukin (IL)-6), and leukocyte rolling, extravasation and adhesion (vascular cell adhesion molecule (VCAM)-1, E-selectin (SELE)) [16]
ROS mediates 4-HPR-induced posttranscriptional expression of the Gadd153 gene
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![PDF] Catalase and glutathione peroxidase are equally active in detoxification of hydrogen peroxide in human erythrocytes](https://figures.semanticscholar.org/e57b719abf8498181e0be7902735507a49da0e4f/2-Figure1-1.png)
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