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The researchers found that rats subjected to CSD displayed a significant decline in erectile function despite no change in serum testosterone levels compared to controls [30]
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Our data suggest that hypermethylation of the 25-hydroxylases, 1--hydroxylase enzymes, and the vitamin D receptor promoter, accompanied by hypomethylation of CYP24A1, leads to impaired expression and possibly a predisposition to VD deficiency/inadequacy
This is intriguing but precision in interpretation matters
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