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The authors concluded that GHK-Cu accelerates wound healing and skin repair through coordinated effects on collagen and glycosaminoglycan turnover, modulation of metalloproteinase activity, attraction of immune and endothelial cells to injury sites, and restoration of replicative vitality in irradiated fibroblasts [1]

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6 The combination of biomechanical and histological endpoints in this model strengthens the mechanistic interpretation: the structural improvement was accompanied by the expected tissue-level changes predicted by the angiogenic and FAK-paxillin mechanisms

The report offers aggregate findings, benchmarks, and guidance for adaptation