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Neurologically, ALD/AMN can manifest with highly variable phenotypes, ranging from very aggressive and rapidly fatal inflammatory CNS demyelination in young boys to a more slowly progressive adult form either with cerebral white matter inflammatory lesions, or a more indolent progressive myelopathy and peripheral neuropathy (Moser, 1997)

As individuals age, the capacity for glutathione synthesis naturally declines, creating a progressive deficit that may contribute to age-related cellular dysfunction and disease susceptibility

Protect from light at all times the copper complex is photosensitive

[DOI] [PubMed] [Google Scholar] 21.McCall WD, Tanner KD, Levine JD