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doi: 10.1038/s41416-019-0451-4

Substitution of bridge carbons for sulphur in calix 4 arene-bis-alpha-hydroxymethylphosphonic acid transformed mobile carrier into ionic channel accompanied with evoked muscle contraction and impaired neurotransmission powered by membrane action of resulting thiocalix 4 arene-bis-alpha-hydroxymethylphosphonic acid

Seven of these studies had an experimental focus and evaluated the application of innovative therapies in the treatment of PCa

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Despite of these findings, most findings regarding the neuroprotective effects of TRZ are obtained from preclinical studies that have not been completely translated into clinical settings