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Given that the glucose-lowering effect of metformin is so strong and is independent of incretin signalling, it might be difficult to detect the beneficial effect of metformin on the incretin axis in human studies

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This transition involves upregulation of markers such as CD206, Arg1, and IL-10, enhanced efferocytosis of apoptotic cells, and secretion of growth factors like VEGF, TGF-, and FGF2, which promote angiogenesis, fibroblast activation, and ECM remodeling [41]

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This effect was attributed to two colonic fermentation processes: the effective release of introduced acyl groups [6] and the microbial generation of additional SCFAs [6, 99]