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Consequently, GLP1R has been a target for developing drugs usually referred to as GLP1R agonists to treat diabetes

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Our signaling-defective mutant integrin inhibits the activation of the cytoplasmic tyrosine kinases FAK and Src, as well as the extracellular signal-regulated kinase, ERK [17, 24]

Altered tissue mercury distribution at 3 hours but showed no changes at 24 hours posttreatment