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Glutamatergic theories of schizophrenia are based on the ability of N-methyl-D-aspartate receptor (NMDAR) antagonists to induce schizophrenia-like symptoms, as well as emergent literature documenting disturbances of NMDAR-related gene expression and metabolic pathways in schizophrenia

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These pathways reduce lipid peroxidation, remodel lipid membranes, and prevent cell ferroptosis through direct or indirect means ( Compared to healthy individuals, RA patients exhibit lower expression of ACSL4 and increased levels of ferritin heavy chain 1 (FTH1), GPX4, and SLC7A11 in the synovium and FLS

Cramer, S

Neural Regener