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GSH performs its protective role with the help of two enzymes, glutathione peroxidase and glutathione reductase [1] (Figure 1)
In the majority of OAD the organic acid accumulates due to deficiency in the mitochondrial catabolism of various CoA-activated carbonic acids such as propionyl-CoA, methylmalonyl-CoA, isovaleryl-CoA, and glutaryl-CoA
Food tracking, even temporarily, can reveal whether dietary drift has occurred
Maintaining the physiological effect on GH release is critical in affecting the maximum concentration of the hormone in the blood without adverse reaction or increased side effects
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