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Early research suggests that MOTS-C peptide may influence pathways such as AMPK activation and antioxidant gene expression, hinting at potential roles in metabolic health, physical performance, and age-related decline

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The downstream effect: increased mitochondrial NAD+ levels, which activate sirtuins (particularly SIRT1 and SIRT3) and AMPK signaling

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It is thought that over time, the disease shifts from an early hypermetabolic state to a hypometabolic state with decreased metabolites (251) and reduced energy production (59), correlating with patient clinical profiles (185)