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The 1.5-3x baseline IGF-1 elevation in trial reports is the pharmacologic mechanism, not an adverse event per se but it informs the monitoring pattern community sources commonly describe
A number of mechanisms appear to contribute to the mucolytic and mucoregulatory benefits of NAC, including directly breaking disulphide bonds between the molecules forming the mucus with its thiol groups and reducing mucus production through its antioxidant effects (oxidative stress has been shown to increase mucus production)
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