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N-acetyl cysteine overdose induced acute toxicity and hepatic microvesicular steatosis by disrupting gsh and interfering lipid metabolisms in normal mice

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A specific oxidoreductase, previously known as apoptosis-inducing-factor mitochondrial-2 (AIFM2), capable of recycling reduced ubiquinol (Co-enzymeQ 10 H 2 ) from ubiquinone at the expense of NAD(P)H, has been presented as a potential ferroptosis inhibitor due to the fact that its overexpression complements the loss of GPx4 in PFA1 and human fibrosarcoma (66, 67)

However, the effect varies, is not guaranteed, and the evidence is still limited

PMID 26313612