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Upon activation, HSF1 trimers translocate to the nucleus and bind to heat shock elements (HSEs) in the promoters of target genes, thereby increasing the transcription of HSP70s, which then undergo degradation or re-monomerization (Vihervaara and Sistonen, 2014)

Additionally, combining such approaches with antioxidants or ferroptosis inhibitors might synergize in protecting chondrocytes and synovial cells from iron-induced damage (Jiang et al., 2021)

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Aberrant activation of STAT3, triggered by cytokines drives the survival, proliferation, and differentiation of MDSCs while upregulating immunosuppressive molecules